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  Indian J Med Microbiol
 

Figure 1: Alternation cell proliferation and apoptosis, alkaline phosphatase, collagen type I, and content of osteoblast by AS treatment (*P < 0.05, vs. control group). (a) Astragaloside dose dependently upregulated cell viability compared with controls. (b) AS dose dependently downregulated cell apoptosis compared with controls. (c) Alkaline phosphatase activity showed a stepwise increase as the concentration of astragaloside treatment increased when compared with controls. (d) Collagen type I was dose dependently upregulated in cell treated with astragaloside. (e) Content of osteoblast treated with various dose of as showed a stepwise increase compared with controls

Figure 1: Alternation cell proliferation and apoptosis, alkaline phosphatase, collagen type I, and content of osteoblast by AS treatment (*<i>P</i> < 0.05, vs. control group). (a) Astragaloside dose dependently upregulated cell viability compared with controls. (b) AS dose dependently downregulated cell apoptosis compared with controls. (c) Alkaline phosphatase activity showed a stepwise increase as the concentration of astragaloside treatment increased when compared with controls. (d) Collagen type I was dose dependently upregulated in cell treated with astragaloside. (e) Content of osteoblast treated with various dose of as showed a stepwise increase compared with controls